Research Paper Volume 12, Issue 14 pp 14174—14188

LncRNA NEAT1/miR-129/Bcl-2 signaling axis contributes to HDAC inhibitor tolerance in nasopharyngeal cancer

Figure 6. miR-129 mediated SAHA tolerance in vivo. (A) The tumor growth curves of nude mice xenografted with C666-1R cells and treated with SAHA and/or miR-129 mimic. (B) The respective tumors. (C) The expression of indicated proteins in different groups of tumors. (D) The mRNA level of Bcl-2 in tumors. (E) The TUNEL staining of tumors in each group. *, p<0.05; **, p<0.01.