LKB1 inactivation leads to centromere defects and genome instability via p53-dependent upregulation of survivin

Li-Yan Jin; Kui Zhao; Long-Jiang Xu; Rui-Xun Zhao; Kaitlin D. Werle; Yong Wang; Xiao-Long Liu; Qiu Chen; Zhuo-Jun Wu; Ke Zhang; Ying Zhao; Guo-Qin Jiang; Feng-Mei Cui; Zhi-Xiang Xu

doi:doi:10.18632/aging.103473

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Research Paper Volume 12, Issue 14 pp 14341—14354

LKB1 inactivation leads to centromere defects and genome instability via p53-dependent upregulation of survivin

Figure 5. Survivin expression is increased in intestinal PJS polyps. (A) HE staining of an intestinal polyp from a PJS patient. (B) IHC detection of survivin expression in intestinal villi and crypts from PJS and control samples.