Research Paper Volume 13, Issue 7 pp 9522—9541

EGB761 ameliorates chronic cerebral hypoperfusion-induced cognitive dysfunction and synaptic plasticity impairment

EGB761 could improve inhibition of mTor signaling pathway after CCH. p-mTor, t-mTor, p-p70S6K, p70S6K, p-4EBP1, 4EBP1, p-eEF2, eEF2, and β-actin in brain homogenates were assayed by Western blotting (A–E) (Con: n=4; 2VO: n=5; 2VO+EGB761: n=6; EGB761: n=5).

Figure 8. EGB761 could improve inhibition of mTor signaling pathway after CCH. p-mTor, t-mTor, p-p70S6K, p70S6K, p-4EBP1, 4EBP1, p-eEF2, eEF2, and β-actin in brain homogenates were assayed by Western blotting (AE) (Con: n=4; 2VO: n=5; 2VO+EGB761: n=6; EGB761: n=5).