Research Paper Volume 13, Issue 19 pp 23036—23071

Low expression of endoplasmic reticulum stress-related gene SERP1 is associated with poor prognosis and immune infiltration in skin cutaneous melanoma

Top 15 enrichment plots of GSEA. The GSEA results showed that DEGs involved in (A) Reactome GPCR ligand binding, (B) Reactome G alpha i signalling events, (C) Reactome class a1 rhodopsin like receptors, (D) Reactome leishmania infection, (E) Naba core matrisome, (F) Kegg neuroactive ligand receptor interaction, (G) Kegg cytokine receptor interaction, (H) Wp GPCRs class a rhodopsin-like, (I) Reactome anti-inflammatory response favouring leishmania parasite infection, (J) Reactome G alpha q signalling events, (K) Kegg chemokine signaling pathway, (L) Reactome peptide ligand binding receptors, (M) Reactome cell surface interactions at the vascular wall, (N) Reactome FC epsilon receptor (FCERI) signaling and (O) Reactome immunoregulatory interactions between a lymphoid and a nonlymphoid cell.

Figure 4. Top 15 enrichment plots of GSEA. The GSEA results showed that DEGs involved in (A) Reactome GPCR ligand binding, (B) Reactome G alpha i signalling events, (C) Reactome class a1 rhodopsin like receptors, (D) Reactome leishmania infection, (E) Naba core matrisome, (F) Kegg neuroactive ligand receptor interaction, (G) Kegg cytokine receptor interaction, (H) Wp GPCRs class a rhodopsin-like, (I) Reactome anti-inflammatory response favouring leishmania parasite infection, (J) Reactome G alpha q signalling events, (K) Kegg chemokine signaling pathway, (L) Reactome peptide ligand binding receptors, (M) Reactome cell surface interactions at the vascular wall, (N) Reactome FC epsilon receptor (FCERI) signaling and (O) Reactome immunoregulatory interactions between a lymphoid and a nonlymphoid cell.