Research Paper Volume 14, Issue 20 pp 8486—8497
The regulatory mechanism of HSP70 in endoplasmic reticulum stress in pepsin-treated laryngeal epithelium cells and laryngeal cancer cells
Figure 1. Pepsin inhibited viability while promoted apoptosis and autophagy in laryngeal epithelial cells and LC cells. (A and B) CCK8 detected the viability in laryngeal epithelial cells and LC cells. (C and D) Apoptosis of laryngeal epithelial cells and LC cells were detected by flow cytometry. (E and F) The autophagy detection kit measured autophagy levels. *p < 0.05, **p < 0.01, ***p < 0.001 vs. Control.