Research Paper Volume 15, Issue 21 pp 11875—11890
MicroRNA-30a-3p: a potential noncoding RNA target for the treatment of arteriosclerosis obliterans
Figure 3. The VSMC phenotypic switch was suppressed by microRNA-30a-3p. (A) PDGF-BB stimulation promoted VSMC phenotypic switching from contractile conditions to synthetic conditions, and microRNA-30a-3p upregulation suppressed this process. (B–D) MicroRNA-30a-3p suppressed VSMC phenotypic protein (SM-α-actin and calponin) switching. **=0.005, ***=0.001.