This article has been corrected: The authors requested to replace Figure 4E and Figure 6. The mistakes of these figures are described below:

Figure 4E: The authors submitted wrong c-kit in panel E of Figure 4.

Figure 6: The order in panels D and E of Figure 6 was reversed. The H2O2 and the H2O2+Res group in the original Figure 6D of apoptotic analysis was reversed.

These corrections do not change any of the conclusions of the publication. The corrected Figure 4E and Figure 6 are provided below.

Morphology and characteristics of OSCs. (E) Reverse transcription PCR analysis for the expression profile of OSCs using ovarian tissue as a positive control. M: 100 bp DNA marker; No RT, PCR of RNA sample without reverse transcriptase.

Figure 4. Morphology and characteristics of OSCs. (E) Reverse transcription PCR analysis for the expression profile of OSCs using ovarian tissue as a positive control. M: 100 bp DNA marker; No RT, PCR of RNA sample without reverse transcriptase.

Resveratrol attenuated H2O2‐induced cytotoxicity and oxidant stress injury in OSCs. (A) CCK8 assay for treated OSCs;*p B) Analysis of intracellular ROS by cell flow cytometry. (C) Immunofluorescence staining of γ‐H2AX and Hoechst. Scale bar: 50 μm. (D) The flow cytometry apoptotic analysis of treated OSCs. (E) Western blotting of related protein expression levels in treated OSCs.

Figure 6. Resveratrol attenuated H2O2‐induced cytotoxicity and oxidant stress injury in OSCs. (A) CCK8 assay for treated OSCs;*p < 0.05. (B) Analysis of intracellular ROS by cell flow cytometry. (C) Immunofluorescence staining of γ‐H2AX and Hoechst. Scale bar: 50 μm. (D) The flow cytometry apoptotic analysis of treated OSCs. (E) Western blotting of related protein expression levels in treated OSCs.