Editorial Volume 11, Issue 5 pp 1319—1320

Figure 1. Lineage-specific cardiac progenitor cells. Overall, our results suggest that the aging and pathological damage of Bmi1⁺ cardiac progenitors could play an important role in heart aging, exacerbating certain pathological responses. Stimulation of endogenous Bmi1⁺ cardiac progenitor cells in the infarcted myocardium would help counteract the pathological remodeling by sustaining injury-induced neovascularization.