Research Paper Volume 12, Issue 10 pp 9604—9620

KMT2A regulates cervical cancer cell growth through targeting VDAC1

Figure 4. KMT2A regulated cervical cancer cell proliferation and colony formation by targeting VDAC1. Human cervical cancer Siha and Caski cells were transfected with KMT2A shRNAs or KMT2A shRNA + VDAC1 overexpression plasmid. At 48 hours after transfection, the cell viability, colony formation and migration ability were measured. (A) Viable Siha and Caski cells. (B) The average cell viability %. (C) Colony formation of Siha and Caski cells. (D) The average count numbers.