Research Paper Advance Articles

Glioma stem cell-derived exosomal miR-944 reduces glioma growth and angiogenesis by inhibiting AKT/ERK signaling

A schematic model shows mechanistic role of miR-944 in glioma angiogenesis. Exosomal miR-944 derived from GSCs reduces angiogenesis of HUVECs by inhibiting VEGFC expression and suppressing the activation of Akt/ERK signaling pathways.

Figure 9. A schematic model shows mechanistic role of miR-944 in glioma angiogenesis. Exosomal miR-944 derived from GSCs reduces angiogenesis of HUVECs by inhibiting VEGFC expression and suppressing the activation of Akt/ERK signaling pathways.