Correction Volume 13, Issue 16 pp 20853

Correction for: TGF-β and NF-κB signaling pathway crosstalk potentiates corneal epithelial senescence through an RNA stress response

NF-κB p65-konckdown attenuates TGF-β1 induced senescence and SASP. (A-B) SA-β-Gal activity and the percentage of SA-β-gal-positive cells in HCECs treated with TGF-β1 for 3 days alone, or in combination with indicated siRNA. (C) Western blot analysis of p16 and p21 in HCECs treated with TGF-β1 for 3 days alone, or in combination with indicated siRNA. (D) The IL-6 and IL-8 in cultured HCECs supernatants were detected by ELISA. **P≤0.01. Data are representative of three independent experiments.

Figure 6. NF-κB p65-konckdown attenuates TGF-β1 induced senescence and SASP. (A-B) SA-β-Gal activity and the percentage of SA-β-gal-positive cells in HCECs treated with TGF-β1 for 3 days alone, or in combination with indicated siRNA. (C) Western blot analysis of p16 and p21 in HCECs treated with TGF-β1 for 3 days alone, or in combination with indicated siRNA. (D) The IL-6 and IL-8 in cultured HCECs supernatants were detected by ELISA. **P≤0.01. Data are representative of three independent experiments.