Commentary Volume 9, Issue 8 pp 1865—1866

Dysfunctional macrophages in Alzheimer Disease: another piece of the “macroph-aging” puzzle?

Laura Costarelli1, , Marco Malavolta1, , Robertina Giacconi1, , Mauro Provinciali1, ,

  • 1 Advanced Technology Center for Aging Research, Scientific and Technological Pole, Italian National Institute of Health and Science on Aging (INRCA), Ancona, Italy

Received: July 31, 2017       Published: August 5, 2017      

https://doi.org/10.18632/aging.101276
How to Cite

Commentary on: Brandon M. Hall, Vitaly Balan, Anatoli S. Gleiberman, Evguenia Strom, Peter Krasnov, Lauren P. Virtuoso, Elena Rydkina, Slavoljub Vujcic, Karina Balan, Ilya Gitlin, Katerina Leonova, Alexander Polinsky, Olga B. Chernova, Andrei V. Gudkov. Aging of mice is associated with p16(Ink4a)- and β-galactosidase-positive macrophage accumulation that can be induced in young mice by senescent cells. Aging. 2016; 8:1294‐1315
10.18632/aging.100991

Copyright: © 2017 Costarelli et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.