Research Paper Volume 10, Issue 10 pp 2695—2708

Role of alternative splicing of VEGF-A in the development of atherosclerosis

Figure 7. Schematic of the model. SRPK1-mediated alternative splicing of VEGF-A to pro-angiogenic VEGF165 may contribute to the development of AS, possibly through increasing AEC proliferation and macrophage apoptosis.