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Research Paper|Volume 10, Issue 10|pp. 2695–2708
Role of alternative splicing of VEGF-A in the development of atherosclerosis
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Figure 7
Figure 7.Schematic of the model. SRPK1-mediated alternative splicing of VEGF-A to pro-angiogenic VEGF165 may contribute to the development of AS, possibly through increasing AEC proliferation and macrophage apoptosis.