Research Paper Volume 13, Issue 6 pp 8026—8039

mTOR inhibition improves mitochondria function/biogenesis and delays cardiovascular aging in kidney transplant recipients with chronic graft dysfunction

(A) Trolox equivalent antioxidant capacity (TEAC) levels in blood from patients treated with calcineurin inhibitor (CNi) or CNi + mTOR inhibitor (CNi+mTORi). (B) Glutathione peroxidase (GPx) activity in peripheral blood mononuclear cells (PBMC) from patients treated with CNi or CNi+mTORi. Statistical difference was assessed by unpaired student’s t-test.

Figure 6. (A) Trolox equivalent antioxidant capacity (TEAC) levels in blood from patients treated with calcineurin inhibitor (CNi) or CNi + mTOR inhibitor (CNi+mTORi). (B) Glutathione peroxidase (GPx) activity in peripheral blood mononuclear cells (PBMC) from patients treated with CNi or CNi+mTORi. Statistical difference was assessed by unpaired student’s t-test.