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Editorial Volume 12, Issue 3 pp 2028—2029

Resilience, disease and the age of single cell science

Michael Simons1,2, , Pei-Yu Chen1, , Thomas W. Chittenden3,4, ,

  • 1 Yale Cardiovascular Research Center, New Haven, CT 06510, USA
  • 2 Department of Cell Biology, Yale University School of Medicine, New Haven, CT 06510, USA
  • 3 WuXi NextCode, Inc., Cambridge, MA 02142, USA
  • 4 Divison of Genetics and Genomics, Boston Children’s Hospital, Harvard Medical School, Boston, MA 02115, USA

Received: November 12, 2019       Published: February 10, 2020      

https://doi.org/10.18632/aging.102850
How to Cite

Copyright: © 2020 Simons et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Corresponding Author

Michael Simons
michael.simons@yale.edu

Keywords
atherosclerosis EndMT aging inflammation artificial intelligence endothelial-to-mesenchymal transition