Research Paper Volume 12, Issue 19 pp 19254—19272
S-allyl cysteine reduces osteoarthritis pathology in the tert-butyl hydroperoxide-treated chondrocytes and the destabilization of the medial meniscus model mice via the Nrf2 signaling pathway
Schematic diagram shows the potential protective effects of SAC on OA. S-allyl cysteine suppresses senescence and apoptosis of chondrocytes and attenuates extracellular matrix metabolic dysfunction in the osteoarthritis model mice via Nrf2.