Research Paper Volume 13, Issue 16 pp 20218—20228

ERCC6L promotes cell growth and metastasis in gastric cancer through activating NF-κB signaling

The effects of ERCC6L overexpression or knockdown on the EMT and NF-κB signaling-related markers in GC cells. (A) qRT-PCR detection of relative ERCC6L mRNA expression in human GC cell lines and a normal gastric epithelial cell line. (B) Western blot analysis of ERCC6L protein expression in human GC cell lines and a normal gastric epithelial cell line. (C) Western blot analysis of the effects of ERCC6L overexpression or knockdown on the EMT and NF-κB signaling-related markers in GC cells. *P

Figure 2. The effects of ERCC6L overexpression or knockdown on the EMT and NF-κB signaling-related markers in GC cells. (A) qRT-PCR detection of relative ERCC6L mRNA expression in human GC cell lines and a normal gastric epithelial cell line. (B) Western blot analysis of ERCC6L protein expression in human GC cell lines and a normal gastric epithelial cell line. (C) Western blot analysis of the effects of ERCC6L overexpression or knockdown on the EMT and NF-κB signaling-related markers in GC cells. *P < 0.05.