Research Paper Volume 13, Issue 16 pp 20218—20228

ERCC6L promotes cell growth and metastasis in gastric cancer through activating NF-κB signaling

The effects of ERCC6L overexpression or knockdown on GC cells growth in vivo. (A) Representative images of the dissected xenograft tumors. Tumor volume was measured every 5 days. Tumors were weighed at the endpoint (30 days). (B) Western blot analysis of the levels of ERCC6L, p-NF-κB p65, E-cadherin and N-cadherin in the dissected xenograft tumors. (n = 6, each group). *P

Figure 4. The effects of ERCC6L overexpression or knockdown on GC cells growth in vivo. (A) Representative images of the dissected xenograft tumors. Tumor volume was measured every 5 days. Tumors were weighed at the endpoint (30 days). (B) Western blot analysis of the levels of ERCC6L, p-NF-κB p65, E-cadherin and N-cadherin in the dissected xenograft tumors. (n = 6, each group). *P < 0.05.