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Special Collection on Mikhail 'Misha' Blagosklonny's Publications in Aging (Albany NY)


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Targeted cancer therapy: the initial high concentration may slow down the selection for resistance

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.206046 pp 11122—11124
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Cellular senescence: when growth stimulation meets cell cycle arrest

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.204543 pp 905—913
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Are menopause, aging and prostate cancer diseases?

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.204499 pp 298—307
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Hallmarks of cancer and hallmarks of aging

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.204082 pp 4176—4187
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As predicted by hyperfunction theory, rapamycin treatment during development extends lifespan

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.203937 pp 2020—2024
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DNA- and telomere-damage does not limit lifespan: evidence from rapamycin

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.202674 pp 3167—3175
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COVID-19
From causes of aging to death from COVID-19

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.103493 pp 10004—10021
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Rapamycin for longevity: opinion article

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.102355 pp 8048—8067
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Rapamycin for the aging skin

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.102664 pp 12822—12826
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Paradoxes of senolytics

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.101750 pp 4289—4293
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Disease or not, aging is easily treatable

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.101647 pp 3067—3078
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Gerosuppression by pan-mTOR inhibitors

Olga V. Leontieva, Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.101155 pp 3535—3551
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Gerosuppression in confluent cells

Olga V. Leontieva, Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100714 pp 1010—1018
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Rapamycin extends life- and health span because it slows aging

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100591 pp 592—598
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M(o)TOR of aging: MTOR as a universal molecular hypothalamus

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100580 pp 490—494
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Rapamycin suppresses brain aging in senescence-accelerated OXYS rats

Nataliya G. Kolosova, Anton O. Vitovtov, Natalia A Muraleva, Andrey E. Akulov, Natalia A. Stefanova, Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100573 pp 474—484
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Big mice die young but large animals live longer

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100551 pp 227—233
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Mechanistic or mammalian target of rapamycin (mTOR) may determine robustness in young male mice at the cost of accelerated aging

Olga V. Leontieva, Geraldine M. Paszkiewicz, Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100528 pp 899—916
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Answering the ultimate question “What is the Proximal Cause of Aging?”

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100525 pp 861—877
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Rapamycin extends lifespan and delays tumorigenesis in heterozygous p53+/− mice

Elena A. Komarova, Marina P. Antoch, Liliya R. Novototskaya, Olga B. Chernova, Geraldine Paszkiewicz, Olga V. Leontieva, Mikhail V. Blagosklonny, Andrei V. Gudkov,
https://doi.org/10.18632/aging.100498 pp 709—714
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New nanoformulation of rapamycin Rapatar extends lifespan in homozygous p53−/− mice by delaying carcinogenesis

Maria Comas, Ilia Toshkov, Karen K. Kuropatwinski, Olga B. Chernova, Alexander Polinsky, Mikhail V. Blagosklonny, Andrei V. Gudkov, Marina P. Antoch,
https://doi.org/10.18632/aging.100496 pp 715—722
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How to save Medicare: the anti-aging remedy

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100479 pp 547—552
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Tumor suppression by p53 without apoptosis and senescence: conundrum or rapalog-like gerosuppression?

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100475 pp 450—455
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Once again on rapamycin-induced insulin resistance and longevity: despite of or owing to

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100461 pp 350—358
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The second international conference "genetics of aging and longevity"

Vladimir N. Anisimov, Andrzej Bartke, Nir Barzilai, Mikhail A. Batin, Mikhail V. Blagosklonny, Holly Brown‐Borg, Yelena Budovskaya, Judith Campisi, Bertrand Friguet, Vadim Fraifeld, Claudio Franceschi, David Gems, Vadim Gladyshev, Vera Gorbunova, Andrei V. Gudkov, Brian Kennedy, Maria Konovalenko, Brian Kraemer, Alexey Moskalev, Isabelle Petropoulos, Elena Pasyukova, Suresh Rattan, Blanka Rogina, Andrei Seluanov, Mikhail Shaposhnikov, Robert Shmookler Reis, Nektarios Tavernarakis, Jan Vijg, Anatoli Yashin, Piotr Zimniak,
https://doi.org/10.18632/aging.100458 pp 305—317
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Cell cycle arrest is not yet senescence, which is not just cell cycle arrest: terminology for TOR-driven aging

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100443 pp 159—165
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Molecular damage in cancer: an argument for mTOR-driven aging

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100422 pp 1130—1141
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Hormesis does not make sense except in the light of TOR-driven aging

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100411 pp 1051—1062
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Yeast-like chronological senescence in mammalian cells: phenomenon, mechanism and pharmacological suppression

Olga V. Leontieva, Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100402 pp 1078—1091
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Progeria, rapamycin and normal aging: recent breakthrough

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100352 pp 685—691
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Cell cycle arrest is not senescence

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100281 pp 94—101
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DNA damaging agents and p53 do not cause senescence in quiescent cells, while consecutive re-activation of mTOR is associated with conversion to senescence

Olga V. Leontieva, Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100265 pp 924—935
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Metformin and sex: why suppression of aging may be harmful to young male mice

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100264 pp 897—899
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Why the disposable soma theory cannot explain why women live longer and why we age

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100253 pp 884—887
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The choice between p53-induced senescence and quiescence is determined in part by the mTOR pathway

Lioubov G. Korotchkina, Olga V. Leontieva, Elena I. Bukreeva, Zoya N. Demidenko, Andrei V. Gudkov, Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100160 pp 344—352
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Why men age faster but reproduce longer than women: mTOR and evolutionary perspectives

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100149 pp 265—273
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Why human lifespan is rapidly increasing: solving "longevity riddle" with "revealed-slow-aging" hypothesis

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100139 pp 177—182
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Impact papers on aging in 2009

Mikhail V. Blagosklonny, Judy Campisi, David A. Sinclair, Andrzej Bartke, Maria A. Blasco, William M. Bonner, Vilhelm A. Bohr, Robert M. Brosh Jr, Anne Brunet, Ronald A. DePinho, Lawrence A. Donehower, Caleb E. Finch, Toren Finkel, Myriam Gorospe, Andrei V. Gudkov, Michael N. Hall, Siegfried Hekimi, Stephen L. Helfand, Jan Karlseder, Cynthia Kenyon, Guido Kroemer, Valter Longo, Andre Nussenzweig, Heinz D. Osiewacz, Daniel S. Peeper, Thomas A. Rando, K Lenhard Rudolph, Paolo Sassone-Corsi, Manuel Serrano, Norman E. Sharpless, Vladimir P. Skulachev, Jonathan L. Tilly, John Tower, Eric Verdin, Jan Vijg,
https://doi.org/10.18632/aging.100132 pp 111—121
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Quantifying pharmacologic suppression of cellular senescence: prevention of cellular hypertrophy versus preservation of proliferative potential

Zoya N. Demidenko, Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100115 pp 1008—1016
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Inhibition of S6K by resveratrol: In search of the purpose

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100059 pp 511—514
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Growth and aging: a common molecular mechanism

Mikhail V. Blagosklonny, Michael N. Hall,
https://doi.org/10.18632/aging.100040 pp 357—362
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Validation of anti-aging drugs by treating age-related diseases

Mikhail V. Blagosklonny,
https://doi.org/10.18632/aging.100034 pp 281—288
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Aging: past, present and future

Mikhail V. Blagosklonny, Judith Campisi, David A. Sinclair,
https://doi.org/10.18632/aging.100009 pp 1—5
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Mikhail ‘Misha’ Blagosklonny’s enduring legacy in geroscience: the hyperfunction theory and the therapeutic potential of rapamycin

David A. Barzilai,
https://doi.org/10.18632/aging.206189 pp 1—15
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